Preliminary experiments point out conolidine could inhibit certain ion channels, cutting down neuronal excitability and restricting agony alerts. This mechanism is especially suitable in neuropathic discomfort, in which abnormal signaling triggers persistent distress. On top of that, conolidine seems to affect G protein-coupled receptor (GPCR) pathways integral to ache https://torreyd481vxk6.tusblogos.com/profile